Laryngomalacia : all you want to know#

What is laryngomalacia,Etiology of laryngomalacia, Common things, Clinical Presentation & Evaluation, management.

Noisy breathing ( stridor) , audible wheeze, change with position, spontaneous resolution. ( laryngomalacia)

Laryngomalacia

Prolapse of the supraglottic structures into the laryngeal airway on inspiration.

Etiology of laryngomalacia



Etiology is not clear, possibly due to incomplete integration of laryngeal sensation with brain stem–mediated reflexes.

Common things with laryngomalacia .


Most of children with laryngomalacia have esophageal and laryngopharyngeal reflux.

Most common congenital anomaly of the upper airway.


Reflux of gastric contents causes inflammation and edema of the laryngeal mucosa, which can worsen laryngomalacia.

Most frequent congenital cause of stridor in infants

Clinical Presentation of laryngomalacia

Stridor is most pronounced at 2 to 4 months of life

Stridor developing in the first 2 weeks of life (not present at birth)

Despite stridor, most children are nontoxic-appearing and in no distress.

▪Worse with feeding, when lying supine, and during periods of agitation


Stridor often changes with position.

Improved with lying prone.

Hoarseness is not a symptom of laryngomalacia and may be suggestive of a vocal cord abnormality.

Some of the patients with laryngomalacia have severe laryngomalacia resulting in respiratory distress, failure to thrive, cyanotic episodes, acute life-threatening events, or an inability to tolerate oral feeds; these patients should be evaluated for concurrent airway lesions .

Laryngomalacia
Laryngomalacia highlights

Diagnosis and Evaluation


Diagnosis is confirmed by direct visualization on flexible transnasal fiber-optic laryngoscopy


Laryngoscopy can be performed while the patient is awake, at bedside, or in the clinic


Findings of laryngomalacia on laryngoscopy.

Short, vertical aryepiglottic folds


Omega-shaped epiglottis, which may be retroflexed



Redundant arytenoid mucosa


Prolapse of redundant tissue overlying the arytenoid cartilage


May be inflammation and edema of laryngeal mucosa, secondary to reflux

Treatment


Most often self-resolves between the ages of 12 and 18 months

Management of gastroesophageal reflux disease (GERD) with histamine 2 blockers or proton pump inhibitors

Children with severe laryngomalacia may require surgical intervention—supraglottoplasty.

In supraglottoplasty, redundant tissue over the arytenoids is removed, and both aryepiglottic folds are divided.

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